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Urticaria and Angioedema
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Your search returned 25 results
from the time period: last 90 days.
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European journal of human genetics : EJHG
Clinical Utility Gene Card for hereditary angioedema with normal C1 inhibitor (HAEnC1)..
Hereditary Angioedema (2), more mentions
... the kinetics of C1-inhibitor (C1-INH) and other complement parameters in a self-limited edematous attack (EA) in a patient with hereditary angioedema due to C1-INH deficiency to better understand the pathomechanism of the evolution, course, and complete resolution of EAs... Keyword: hereditary angioedema with C1-inhibitor deficiency.
Hereditary Angioedema (3), more mentions
Allergy and asthma proceedings
AbstractText: Severe attacks of hereditary angioedema (HAE) are debilitating and potentially life threatening, and can increase anxiety and the use of medical resources AbstractText: This post hoc assessment evaluated recombinant human C1 esterase inhibitor (rhC1-INH) used to treat acute severe HAE attacks AbstractText: In a double-blind, randomized-controlled ...
Hereditary Angioedema (2), more mentions
Allergy and asthma proceedings
... quality of life (HR-QoL) is impaired in patients with hereditary angioedema (HAE) but has not yet been satisfactorily described AbstractText: To ... RAND Corporation Short Form 36 (RAND-36), the disease-specific Angioedema Quality of Life instrument (AE-QoL), the recently introduced Angioedema Activity Score (AAS) form, and questionnaires on sick leave and ...
Angioedema (2), Depressive Disorder (2), Hereditary Angioedema (2), more mentions
PloS one
Attacks of Hereditary Angioedema due to C1-inhibitor deficiency (C1-INH-HAE)are often triggered by stressful events/hormonal changes.Our study evaluates the relationship between autonomic nervous system (ANS) and contact/complement system activation.Twenty-three HAE patients (6 males, mean age 47.5±11.4 years) during remission and 24 ...
Hereditary Angioedema (2), more mentions
Journal of clinical psychopharmacology
Angioedema Associated With Long-Acting Injectable Paliperidone Palmitate: A Case Report..
Angioedema (2), more mentions
Angioedema has been observed in a few cases secondary to systemic lupus erythematosus (SLE. Herein, we report a rare case where a young healthy male initially presented with angioedema, lymphadenopathy and parotitis and later on developed neuropsychiatric manifestations at the very onset of his SLE disease. This case illustrates the importance of prompt clinical consideration of lupus with unusual and ...
Immune System Diseases (3)
Angioedema (3), Parotitis (2), Lymphadenopathy (1), more mentions
... of life (QoL) in chronic spontaneous urticaria (CSU) patients with angioedema refractory to high doses of H1 -antihistamines AbstractText: In X ... placebo-controlled study, CSU patients (18-75 years) with ≥4 angioedema episodes during the 6 months before inclusion were randomized (1 ... was also associated with decreased fear of suffocation due to angioedema AbstractText: Our findings support omalizumab treatment in patients with severe H1-antihistamine-refractory CSU with angioedema ...
Neuroscience (1), Dermatology (1)
Angioedema (8), Urticaria (3), Asphyxia (1), more mentions
The Journal of emergency medicine
... agents for anaphylactic reactions have limited efficacy for bradykinin-mediated angioedema and, therefore, agents approved for hereditary angioedema are increasingly prescribed for these patients AbstractText: This systematic review ... C1-INH), icatibant, and ecallantide for treatment of ACEI-induced angioedema AbstractText: A PubMed search was conducted and articles were cross ... Of the novel agents traditionally used for hereditary angioedema, icatibant has the highest level of evidence and has been reported to be successful in limiting the progression of angioedema Keyword: C1-esterase inhibitor ...
Angioedema (8), Hereditary Angioedema (3), Anaphylaxis (1), more mentions
Journal of the American Academy of Dermatology
No Abstract Available
Urticaria (2), more mentions
Immunology and allergy clinics of North America
Aspirin and nonsteroidal antiinflammatory drugs (NSAIDs) are widely used in the United States and throughout the world for a variety of indications. Several unique hypersensitivity syndromes exist to this class of medications, making them one of the common reasons for consultation to the allergist. The lack of any laboratory-based diagnostic studies to assist in identifying the culprits in these reactions make evaluation of aspirin and NSAID hypersensitivity challenging. Identifying patients appropriate for oral challenge and/or desensitization protocols is the standard pragmatic approach to this issue when it arises.
Muscular and Skeletal Diseases (2), more mentions
Journal of cardiothoracic and vascular anesthesia
No Abstract Available
Urticaria (2), more mentions
The British journal of dermatology
The human body interacts with the environment mainly through three major tissues: the lungs, skin, and gastrointestinal (GI) tract. In each one, an intensive nervous network is present, conjugated with abundant mast cells (MC). While MC-mediated disorders in airways and the skin are extensively discussed, MC-mediated disorders of the GI tract remain relatively uncommon. Since the GI tract is one of the three main environment-interacting tissues, it is reasonable that MC-mediated disorders of the GI may have the same weight and impact as asthma or chronic urticaria (CU). This article is protected by copyright. All rights reserved.
Urticaria (2), Irritable Bowel Syndrome (1), Asthma (1), more mentions
Contact dermatitis
No Abstract Available
Urticaria (2), more mentions
Contact dermatitis
No Abstract Available
Urticaria (2), more mentions
The Journal of allergy and clinical immunology
Histamine and LPS synergistically induce the expression of TF by vascular endothelial cells, activating the extrinsic coagulation cascade followed by an increase of vascular permeability.
Clinical immunology (Orlando, Fla.)
This paper summarizes and reviews the mechanisms of action and data concerning efficacy of recommended treatments as well as other treatments that have been tested, independently of the outcomes, in the management of chronic spontaneous urticaria. Due to the central role of mast cells and histamine in the pathophysiology of this disease, H1-antihistamines remain the first-line treatment. However, current knowledge about this complex disease, also recognizes an important role for T lymphocytes, B lymphocytes, and autoantibodies. Implications of these others mediators thus provide further targets for treatment. Indeed, agents previously used to treat other autoimmune and inflammatory diseases, have demonstrated efficacy in chronic spontaneous urticaria and are therefore potential therapeutic alternatives for antihistamine unresponsive patients.
Immune System Diseases (1)
Urticaria (4), more mentions
... with differential, D-dimer, fibrinogen, C3, C4, IL-6 etc. For most patients, we also collected data on age, gender, duration of CSU, presence of angioedema, activity (UAS at the time of blood sampling and for 7 days), quality of life (CU-Q2oL and/or DLQI), comorbidities and possible causes of ...
Cardiovascular Diseases (1)
Urticaria (4), Angioedema (1), Hypertension (1), more mentions
BACKGROUND: Omalizumab is an effective and well-tolerated treatment for chronic spontaneous urticaria (CSU). Markers and predictors of response are largely unknown, but needed to optimize omalizumab treatment. Omalizumab targets IgE, and IgE levels may be linked to the effects of treatment. We evaluated if response rates to treatment with omalizumab in patients with CSU are linked to their baseline IgE levels, their IgE levels after omalizumab treatment, and the ratio of on treatment IgE and baseline IgE levels. METHODS: CSU patients (n=113) were treated with omalizumab 300 mg/4 weeks for 12 weeks, when their treatment responses, i.e. no, partial, or complete response, were assessed by use of the urticaria activity score, physician and patient visual analog scale, and treatment effectiveness score. Total IgE levels were measured before treatment (bIgE) with omalizumab and 4 weeks thereafter (w4IgE). RESULTS: Non-responders to omalizumab had significantly lower bIgE levels (17.9IU/ml, 17.0-55.0IU/ml) than partial responders (82.0IU/ml, 46.2-126.5IU/ml, p=0.008) and complete responders (73.7IU/ml, 19.45-153.8IU/ml, p=0.032). Non-responders also had lower w4IgE levels and lower ratios of w4IgE/bIgE levels than partial and complete responders (p<0.001). Non-response to omalizumab was best predicted by patients' w4IgE/bIgE ratios, significantly better than by bIgE levels (p=0.016). CONCLUSIONS: In CSU, total IgE levels and their change predict the response to treatment with omalizumab. The assessment of pre- and post-treatment IgE levels and their ratio may help to improve the management of CSU in patients who require omalizumab treatment. This article is protected by copyright. All rights reserved.
Urticaria (3), more mentions
Contact dermatitis
In the dermatological literature, the term 'contact urticaria syndrome' is often used with patients presenting no symptoms of contact urticaria, but symptoms of adjacent mucous membranes evolving to a systemic allergic reaction. Contact urticaria refers to a wheal and flare reaction following external contact with a causative substance. Its use should be restricted to skin: a skin reaction after direct skin contact.
Urticaria (5), Allergy (1), Conjunctivitis (1), more mentions
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